<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2413-4341</journal-id>
<journal-title><![CDATA[Revista Paraguaya de Reumatología]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. parag. reumatol.]]></abbrev-journal-title>
<issn>2413-4341</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Paraguaya de Reumatología]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2413-43412021000200055</article-id>
<article-id pub-id-type="doi">10.18004/rpr/2021.07.02.55</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Efecto de la dieta libre de gluten sobre los niveles de interferón gamma (IFN-&#947;) en pacientes con enfermedad celíaca]]></article-title>
<article-title xml:lang="en"><![CDATA[Effect of gluten-free diet on interferon gamma (IFN-&#947;) levels in patients with celiac disease]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ferreira]]></surname>
<given-names><![CDATA[Silvia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Barúja]]></surname>
<given-names><![CDATA[Jorge]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ortiz]]></surname>
<given-names><![CDATA[Jesús]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Masi]]></surname>
<given-names><![CDATA[José]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Chamorro]]></surname>
<given-names><![CDATA[María Elena]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Carpinelli]]></surname>
<given-names><![CDATA[María Mercedes]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Giménez]]></surname>
<given-names><![CDATA[Vivian]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Langjahr]]></surname>
<given-names><![CDATA[Patricia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional de Asunción Instituto de Investigaciones en Ciencias de la Salud Departamento de Inmunología]]></institution>
<addr-line><![CDATA[San Lorenzo ]]></addr-line>
<country>Paraguay</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Nacional de Asunción Facultad de Ciencias Médicas Hospital de Clínicas, Departamento de Gastroenterología y Endoscopía]]></institution>
<addr-line><![CDATA[San Lorenzo ]]></addr-line>
<country>Paraguay</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Fundación Paraguaya de Celíacos (FUPACEL)  ]]></institution>
<addr-line><![CDATA[Asunción ]]></addr-line>
<country>Paraguay</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2021</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2021</year>
</pub-date>
<volume>7</volume>
<numero>2</numero>
<fpage>55</fpage>
<lpage>59</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.iics.una.py/scielo.php?script=sci_arttext&amp;pid=S2413-43412021000200055&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.iics.una.py/scielo.php?script=sci_abstract&amp;pid=S2413-43412021000200055&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.iics.una.py/scielo.php?script=sci_pdf&amp;pid=S2413-43412021000200055&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen: Introducción: La enfermedad celíaca (EC) es una enteropatía crónica autoinmune, con inflamación principal a nivel del intestino delgado, gatillada por la exposición al gluten de la dieta en individuos genéticamente susceptibles. Las citoquinas inflamatorias se encuentran implicadas en la patogénesis de la EC, entre ellas una citoquina relevante es el interferón gamma (IFN-&#947;). Los niveles de esta citoquina se encuentran alterados en la EC y se relaciona con daños a la mucosa intestinal. La dieta libre de gluten (DLG) es el único tratamiento efectivo para la EC. Objetivo: Determinar el efecto de una DLG sobre los niveles circulantes de IFN-&#947; en pacientes adultos paraguayos con EC. Métodos: Se incluyeron 40 pacientes con EC, de los cuales 20 no presentaban DLG debido a su diagnóstico reciente y 20 presentaban una DLG. Además, se incluyeron 15 sujetos controles aparentemente sanos. Resultados: Los pacientes con EC sin DLG presentaron niveles séricos de IFN-&#947; aumentados respecto al grupo control. Contrariamente, los pacientes con DLG no presentaron diferencias en los niveles de la citoquina al comparar con el grupo control. Conclusión: Los niveles séricos de la citoquina pro-inflamatoria IFN-&#947; se encontraron elevados en los pacientes adultos con EC sin una DLG en comparación a individuos controles, lo cual aporta información sobre el proceso inflamatorio presente en este grupo de pacientes.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract: Introduction: Celiac disease (CD) is a chronic autoimmune enteropathy, with inflammation mainly at the small intestine triggered by exposure to dietary gluten in genetically susceptible individuals. Inflammatory cytokines are implicated in the pathogenesis of CD, among them a relevant cytokine is interferon gamma (IFN-&#947;). Levels of this cytokine are altered in CD and its expression has been linked to intestinal mucosal damage. The gluten-free diet (GFD) is the only effective treatment for CD. Aim: To determine the effect of GFD on circulating levels of IFN-&#947; in adult Paraguayan patients with CD. Methods: 40 patients with CD were included, of which 20 did not present GFD due to recent diagnosis and 20 presented a GFD. In addition, 15 apparently healthy controls were included. Results: Patients with CD without GFD had increased serum IFN-&#947; levels compared to the control group. In contrast, patients with GFD did not show differences in cytokine levels when compared to the control group. Conclusion: Serum levels of the pro-inflammatory cytokine IFN-&#947; were found to be elevated in adult CD patients without a GFD compared to controls, which provides information about the inflammatory process present in this group of patients.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[enfermedad celíaca]]></kwd>
<kwd lng="es"><![CDATA[dieta]]></kwd>
<kwd lng="es"><![CDATA[libre de gluten]]></kwd>
<kwd lng="es"><![CDATA[inflamación]]></kwd>
<kwd lng="es"><![CDATA[citoquinas]]></kwd>
<kwd lng="es"><![CDATA[interferón gamma]]></kwd>
<kwd lng="en"><![CDATA[celiac disease]]></kwd>
<kwd lng="en"><![CDATA[diet]]></kwd>
<kwd lng="en"><![CDATA[gluten-free]]></kwd>
<kwd lng="en"><![CDATA[inflammation]]></kwd>
<kwd lng="en"><![CDATA[cytokines]]></kwd>
<kwd lng="en"><![CDATA[interferon gamma]]></kwd>
</kwd-group>
</article-meta>
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