<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2312-3893</journal-id>
<journal-title><![CDATA[Revista Virtual de la Sociedad Paraguaya de Medicina Interna]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. virtual Soc. Parag. Med. Int.]]></abbrev-journal-title>
<issn>2312-3893</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Paraguaya de Medicina Interna]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2312-38932019000100075</article-id>
<article-id pub-id-type="doi">10.18004/rvspmi/2312-3893/2019.06(01)75-085</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Conceptos epidemiológicos, manejo diagnóstico y farmacológico de la insuficiencia cardiaca congestiva crónica]]></article-title>
<article-title xml:lang="en"><![CDATA[Epidemiological concepts, diagnostic and pharmacological management of chronic congestive heart failure]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Chávez Alfonso]]></surname>
<given-names><![CDATA[Christian Osmar]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Centurión]]></surname>
<given-names><![CDATA[Osmar Antonio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional de Asunción Facultad de Ciencias Médicas Hospital de Clínicas]]></institution>
<addr-line><![CDATA[San Lorenzo ]]></addr-line>
<country>Paraguay</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Sanatorio Metropolitano Departamento de Investigación en Ciencias de la Salud. ]]></institution>
<addr-line><![CDATA[Fernando de la Mora ]]></addr-line>
<country>Paraguay</country>
</aff>
<pub-date pub-type="pub">
<day>01</day>
<month>03</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>01</day>
<month>03</month>
<year>2019</year>
</pub-date>
<volume>6</volume>
<numero>1</numero>
<fpage>75</fpage>
<lpage>85</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.iics.una.py/scielo.php?script=sci_arttext&amp;pid=S2312-38932019000100075&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.iics.una.py/scielo.php?script=sci_abstract&amp;pid=S2312-38932019000100075&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.iics.una.py/scielo.php?script=sci_pdf&amp;pid=S2312-38932019000100075&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN La insuficiencia cardiaca (IC) es un síndrome clínico complejo que resulta de cualquier deterioro estructural o funcional del llenado ventricular o gasto cardiaco. Las manifestaciones clínicas típicas de la insuficiencia cardiaca son disnea y fatiga, que pueden limitar la tolerancia al ejercicio, y retención de líquidos, lo que pueden conducir a la congestión pulmonar y esplácnica, y edema periférico. La prevalencia de la IC se sitúa aproximadamente en el 1-2% de la población adulta en países desarrollados, y aumenta a más del 10% entre personas de 70 o más años de edad. El riesgo de IC a los 55 años es del 33% para los varones y el 28% para las mujeres. Es categórico el hecho de la existencia de una tendencia demográfica hacia el aumento de la longevidad de la población en general, lo cual implica a su vez un aumento en la incidencia de la insuficiencia cardiaca. Por lo tanto, las investigaciones científicas se deben dirigir al estudio del manejo diagnóstico y terapéutico de esta población con edad avanzada. Normalmente, la miocardiopatía es la causa de la disfunción ventricular sistólica o diastólica. No obstante, las anomalías de válvulas, pericardio, endocardio, ritmo cardiaco y conducción también pueden causar IC, en algunas ocasiones se puede deber a la combinación de más de una de ellas. Los objetivos del tratamiento para los pacientes con IC son mejorar el estado clínico, la capacidad funcional y la calidad de vida, prevenir las hospitalizaciones y reducir la mortalidad. Los estudios experimentales y clínicos deben enfocarse a seguir aportando datos sobre la fisiopatología, la influencia de la edad, los criterios diagnósticos, la caracterización de subgrupos especiales, y la identificación de nuevos agentes farmacológicos para mejorar la estrategia terapéutica preventiva y curativa en pacientes con insuficiencia cardiaca congestiva crónica.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT Heart failure (HF) is a complex clinical syndrome that results from any structural or functional deterioration of the ventricular filling or cardiac output. The typical clinical manifestations of heart failure are dyspnea and fatigue, which can limit exercise tolerance, and fluid retention leading to pulmonary and splanchnic congestion, and peripheral edema. The prevalence of HF is approximately 1-2% of the adult population in developed countries, and increases to more than 10% among people 70 years or older. The risk of HF at age 55 is 33% for men and 28% for women. There is a categorical tendency towards an increase in life expectancy in the world population, which consequently results in an increased incidence of heart failure. Therefore, scientific research should focus on searching for better diagnostic and treatment strategies in this elderly population. Frequently, cardiomyopathy is the cause of systolic or diastolic ventricular dysfunction. However, the heart valves diseases, pericardium, endocardium, cardiac conduction and rhythm disorders can also cause HF, in some cases it can be caused by a combination of more than one of them. Treatment goals for patients with HF are aimed on optimizing clinical status, improving functional capacity and quality of life, preventing hospitalizations and reducing mortality. Experimental and clinical studies should focus on continuing to provide data on the pathophysiology of heart failure, on diagnostic criteria, the characterization of special subgroups, and the identification of new pharmacological agents to improve the preventive and curative therapeutic strategy in patients with chronic congestive heart failure.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[insuficiencia cardiaca]]></kwd>
<kwd lng="es"><![CDATA[terapéutica]]></kwd>
<kwd lng="es"><![CDATA[cardiomiopatías]]></kwd>
<kwd lng="es"><![CDATA[volumen sistólico]]></kwd>
<kwd lng="es"><![CDATA[epidemiología]]></kwd>
<kwd lng="en"><![CDATA[heart failure]]></kwd>
<kwd lng="en"><![CDATA[therapeutics]]></kwd>
<kwd lng="en"><![CDATA[cardiomyopathies]]></kwd>
<kwd lng="en"><![CDATA[stroke volume]]></kwd>
<kwd lng="en"><![CDATA[epidemiology]]></kwd>
</kwd-group>
</article-meta>
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